Index
Module 11 • Cardiology
Cardiovascular Critical Care I
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Data Tables
Cardiovascular Critical Care I
Sajni V. Patel ~2 min read Module 11 of 20
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Cardiovascular Critical Care I

Patient Case (Continued)

4

J.M.’s ECG results reveal no acute evidence of ST segment changes. However, the resident is still consider-

ing a diagnosis of ACS, given the patient’s shortness of breath and mild troponin elevation. Which statement

would be most accurate regarding the potential for other potential diagnoses?

A.No, this is most likely an NSTEMI.
B.Yes, it is likely undiagnosed chronic obstructive pulmonary disease.
C.Yes, it is likely early sepsis.
D.Yes, it is likely decompensated HF.
5

J.M. has been experiencing intermittent bradycardia on telemetry. The team has consulted the cardiac elec-

trophysiology team. In the meantime, which statement most accurately reflects whether any other underlying

correctable/contributing causes can be addressed?

A.Ticagrelor could be discontinued and switched back to clopidogrel.
B.Ticagrelor should be discontinued altogether.
C.No treatment intervention is needed; this is likely the result of J.M.’s HF progression.
D.J.M.’s hyperkalemia should be treated.
V.ARRHYTHMIAS AND ANTIARRHYTHMICS
A.Pathophysiology: Arrhythmias are generally caused by 2 main mechanisms:
1

Abnormal impulse formation

Altered automaticity

Abnormal automaticity

Triggered activity—early or delayed

2Abnormal impulse conduction

Reentry

Conduction block

B.Bradyarrhythmias (beyond sinus) and Types of Heart Block (Critical Care Medicine 2014:xix;

Pathophysiology of Heart Disease 2011:xiv; BMJ 2002;324:662-5; BMJ 2002;324:415-8)

1

Etiologies of heart block (Box 3)

Box 3. Causes of Bradyarrhythmias

CAD
Degenerative conduction disease
Drug induced
Electrolyte disturbances (particularly hyperkalemia)
Endocarditis
Myocarditis
Surgery (particularly cardiac surgery)
Tumors
Vagus nerve–mediated heart block

CAD = coronary artery disease.

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