Index
Module 11 • Cardiology
Cardiovascular Critical Care I
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Data Tables
Cardiovascular Critical Care I
Sajni V. Patel ~3 min read Module 11 of 20
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Cardiovascular Critical Care I

Table 5. Parenteral Antithrombotics (Circulation 2014;130:e344-426; Circulation 2013;127:e362-425;Circulation 2015;131:1123-49; J Am Coll Cardiol

2011;58:e123-210; J Am Coll Cardiol 2011;58:e44-122; Br J Clin Pharmacol 2011;72:647-57)

Medication

Heparin (UFH)a

Bivalirudin

Eptifibatide

Tirofiban

Cangrelorb

Mechanism of

action

Indirect thrombin

inhibition (binds to

antithrombin III)

Direct thrombin inhibitor

Binds to GP IIb/IIIa platelet

receptor, inhibiting final

common pathway for platelet

aggregation

Binds to GP IIb/

IIIa platelet receptor,

inhibiting final

common pathway for

platelet aggregation

Inhibits ADP-mediated platelet

activation at P2Y12 receptor

Bolus dose

50–100 units/kg

based on target

activated clotting

time (ACT)

0.75 mg/kg

180 mcg/kg x 2, 10 min apart

25 mcg/kg

30 mcg/kg

Continuous

infusion

Not typically given

during PCI

1.75 mg/kg/hr

2 mcg/kg/min

0.15 mcg/kg/min

4 mcg/kg/min

Platelet inhibition

Indirectly inhibits

thrombin-mediated

platelet activation

Indirectly inhibits thrombin-mediated

platelet activation

Restoration of platelet function

within 6–8 hr of discontinuation

Restoration of

platelet function

within 6–8 hr of

discontinuation

Onset of platelet inhibition

within min of administration

Restoration of platelet function

within 1 hr of discontinuation

Elimination

Hepatic and reticulo-

endothelial system

80% plasma proteolysis

20% renal

Renal (50%)

Renal (65%)

Plasma dephosphorylation

Removed by

dialysis

No

Partial

Yes

Yes

N/A

Other notable

adverse effect(s)

or clinical pearls

Target ACT of about

200–300 s during

procedure based on

presence of GP IIb/

IIIa inhibitor and

device used

Requires renal dose adjustment

Useful in patients with suspected or

confirmed HIT

May be preferred in patients with higher

bleeding risk (particularly compared

with UFH with GP IIb/IIIa inhibitor

and among patients who undergo a

transfemoral PCI approach)

Requires renal dose adjustment

Acute, profound

thrombocytopenia

Requires renal dose

adjustment

Acute, profound

thrombocytopenia

Oral prasugrel and clopidogrel

must be given immediately

after discontinuation of

cangrelor infusion to maintain

platelet inhibition. Ticagrelor

should be given as soon as

possible, and administration

may overlap with cangrelor

infusion to maintain platelet

inhibition after cessation of

cangrelor

aEnoxaparin may be used as an alternative to UFH.

bU.S. Food and Drug Administration (FDA) approved as an adjunct to PCI for reducing the risk of periprocedural myocardial infarction (MI), repeat coronary revascularization, and stent thrombosis (ST) in patients who have

not been treated with a P2Y12 platelet inhibitor and are not receiving a glycoprotein IIb/IIIa inhibitor.

ACT = activated clotting time; GP = glycoprotein; HIT = heparin-induced thrombocytopenia; N/A = not applicable; UFH = unfractionated heparin.

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