Index
Module 10 • Neurology
Neurocritical Care
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Neurocritical Care
Keaton S. Smetana ~3 min read Module 10 of 20
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Neurocritical Care

Combination with mechanical thrombectomy

Rescue therapy in patients having received intravenous thrombolytic

Large hemispheric infarction

Dose is not well defined.

ii.

Typically applied until thrombus has resolved

iii.

Alteplase dose less than 20 mg

Patient Case

Questions 5 and 6 pertain to the following case.

A 49-year-old woman presents to an urgent treatment center with the β€œworst headache of her life.” She is

sent to your emergency department, where a head CT scan reveals a diffuse SAH. The patient takes no home

medications and has an insignificant medical history other than a 20 pack-year history of smoking.

5

Which therapy is most appropriate to prevent ischemic complications from SAH?

A.Nimodipine for 21 days.
B.Euvolemia and permissive hypertension for 14 days.
C.Simvastatin for 14 days.
D.Aminocaproic acid infusion for 48 hours.
6

On hospital day 5, the patient has reduced alertness, and her GCS score decreases by 2 points. The digital

subtraction angiography suggests cerebral vasospasm. Which treatment modality is best to initiate first?

A.Norepinephrine 0.05 mcg/kg/minute to increase MAP to 90 mm Hg.
B.One unit of packed RBCs to increase Hgb to 10 g/dL.
C.3% sodium chloride boluses to increase central venous pressure to 14 mm Hg.
D.Milrinone 0.375-mcg/kg/minute infusion to increase cardiac index to 5 L/minute/m2.
D.Stent Deployment and Antiplatelet Agents
1

Intracranial stents are often deployed in place of coils or to support coils for complex aneurysms.

2Intracranial circulation is different from coronary circulation.

Blood vessels are generally smaller and more tortuous.

Flow rate is lower.

Epithelialization of stent takes longer.

3

Dual antiplatelet therapy is typically used around the time of stent placement.

Clopidogrel (a prodrug) plus aspirin

Current evidence suggests therapy for up to 3 months (not 4 weeks like after percutaneous coronary

intervention), followed by aspirin monotherapy thereafter.

Platelet testing may be necessary in some individuals to evaluate their pharmacogenetic response

to clopidogrel.

d.No gold standard for platelet reactivity testing in this setting. VerifyNow is commonly used and

measures platelet reactivity units (PRU) for aspirin and P2Y12 inhibitors.

Some patients may not respond to clopidogrel (up to 30%), and additional loading doses and

increased maintenance doses are not effective.

Ticagrelor may play a role in patients with a variable response to clopidogrel (must be used in

combination with aspirin less than 100 mg) (Neurocrit Care 2024;40:262-71).

Prasugrel (a prodrug) is not recommended (unless absolutely necessary) in patients with high

on-treatment platelet reactivity (e.g. clopidogrel non-responders) because of warnings about use

in patients with a history of stroke – Increased bleeding. Reduced doses (2.5–5 mg) have been

reported with some success in preventing thrombosis without excessive bleeding.

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