Index
Module 12 • Cardiology
Cardiovascular Critical Care II
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Data Tables
Cardiovascular Critical Care II
Patrick M. Wieruszewski ~3 min read Module 12 of 20
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Cardiovascular Critical Care II

(6)Eclampsia: new-onset tonic-clonic, focal, or multifocal seizures in the absence of

other causative conditions often associated (but not required) with hypertension in

pregnancy

(b)Preeclampsia with severe features can only be managed by delivery of the baby.
(c)Magnesium can be considered as an adjunctive therapy to decrease seizure risk or if

seizures develop.

(d)Intravenous medications should only be considered for (1) severe-range hypertension, (2)

preeclampsia/eclampsia, and/or (3) hypertensive emergency (signs of target organ damage)

in a pregnant patient.

(e)Hydralazine and labetalol are feasible first-line options, and labetalol may have fewer
adverse effects (Am J Health Syst Pharm 2009;66:337-44). Choice of agent should be

dependent on provider familarity and local practices and protocols (Cochrane Database

Syst Rev 2013;2013:CD001449).

(f)BP target goal for severely elevated BPs, preeclampsia/eclampsia, and hypertensive

emergency in a pregnant patient is 160/110 mm Hg or less with avoidance of abrupt

decreases in BP, which can lead to potentially harmful fetal effects. Because of this

caution, the MAP should be decreased by ~20%-25% over the first few minutes to hours,

and the BP should then be further decreased to the target of 160/110 mm Hg or less over

the subsequent hours.

ii.

Catecholamine-induced hypertensive emergency

(a)Phentolamine is the drug of choice because it competitively inhibits Ξ±-adrenergic receptors.
(b)Ξ²-Selective antagonists are contraindicated unless the patient is fully Ξ±-blocked.

iii.

Cocaine-induced hypertensive emergency (Ann Emerg Med 2008;52:S18-20; Chest

2007;131:1949-62)

(a)Benzodiazepines are used to target the central effects of cocaine as first-line therapy and

often will result in control of tachycardia and hypertension. Can consider diazepam 5–10

mg intravenously or lorazepam 2–4 mg intravenously titrated to effect.

(b)If central control of cocaine-induced hypertension fails, consider direct Ξ±-antagonism with

phentolamine.

(1)Phentolamine 1 mg intravenously; repeat every 5 minutes as needed.
(2)If direct Ξ±-antagonism does not gain control or is unavailable, consider additional

antihypertensives:

(A)Nitroglycerin, nicardipine, nitroprusside, or fenoldopam titrated to effect are

viable options (see Tables 9 and 10 for dosing and considerations).

(B)Verapamil and diltiazem decrease coronary vasospasm associated with acute
cocaine intoxication (Am J Cardiol 1994;73:510-3).
(C)Controversy exists regarding the use of Ξ²-blockers.
β€’Labetalol has shown conflicting results regarding ability to control MAP but

does not alleviate cocaine-induced coronary vasoconstriction.

β€’Consensus opinion recommends Ξ²-selective antagonists only if full

Ξ±-antagonism is used first.

iv.

Blood pressure variability (BPV)

(a)Concept of BPV emerging as a key therapeutic target in various populations (Stroke

2014;45:2275-9; Eur J Neurol 2013;20:1277-83; J Cardiothorac Vasc Anesth 2014;28:579-

85).

(b)BPV is often expressed as the standard deviation of SBP, MAP, DBP, or the area under the

curve of time spent outside of blood pressure target.

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